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Consumer
behaviors have been thought that they are influenced by a variety of factors.

Level of involvement of a buying decision is seen as low or high fluctuates by
consumers, not by products. Based on customer’s experience and knowledge, some
buyers are able to make quick buying decisions, while others may need to
acquire more information and evaluate them before jump into a decision. The extent
of involvement indicates the importance of the product, the interest in
consuming it, and the amount of necessary information to close a purchase. The extent
of involvement in purchasing decisions may be seen as a sequence ranging from repetitive
decisions (consumers are indifferent) to high-involved decisions that
necessitate an extensive consideration. Consumers with no idea about a product
may be more involved than somebody already has experienced it. People have
often thought about several products they need or want but stopped at that
stage. Sometimes, they come across those products and look at them, evaluate
and compare them considerately, then stop and never proceed to the purchase
stage. When some products are running out such as milk, sugar, they will be are
refilled right after consumers recognize the need. Regular, basic products do
not necessitate consumers to search for more information or assess other options.

As Nike says in their slogan “just do it”. The level of involvement in the
buying decisions incline to decrease if the products are relatively cheap, and
carry a low risk (both financial and timing) if the buyer are disappointed by
buying them. 

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The extent to which is can be

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The third iteration
of the Dopamine Hypothesis of Schizophrenia makes reference to the theory that schizophrenic
symptoms are caused by pre synaptic hyper dopaminergic signalling due to
potential risk factors (Howes and Kapur, 2009). There is a broad consensus that
the dopamine hypothesis has contributed to a significant understanding of the
underlying mechanisms of schizophrenia due to dopaminergic dysregulation in
specific pre frontal brain regions (Brisch et al., 2014). It should be acknowledged that schizophrenia
is not solely psychotic. Negative and cognitive deficits need be taken into
account in regards to hyper dopaminergic states to comprehend the disorder it its
entirety, as both link to poor quality of life and an inability to function (Lin et al., 2013). Howes and Kapur (2009) acknowledge
that the Dopamine Hypothesis III gravitates towards “psychosis – in –
schizophrenia” (p.556), potentially limiting the extent to which is can be
applied to every aspect of schizophrenia. This essay will consider an array of evidence
to disregard these accusations and acknowledge the extent to which excessive
dopamine affects the onset of schizophrenic symptoms. Albeit there is strong evidence
of the role of dopamine, it fails to acknowledge what may be influencing
abnormal dopamine levels. Glutamate has been found to regulate dopamine
synthesise through NMDAR suggesting dopamine activity may be controlled through
this neurotransmitter. 

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